Within this review, we explore the molecular targets of PEA, its pharmacological activity in neurological disorders, suffering notion and inflammatory ailments, and also the present evidence around the efficiency of micronized and extremely‐micronized formulations of PEA when supplied alone or in combination with the antioxidant flavonoids, such
2016a). This smaller maximize was proportionate into the smaller peak of PEA stages detected in human volunteers in comparison with beagle pet dogs. This difference, consequently, is often attributable to the decreased total quantity of PEA acutely administered to human volunteers (about ∼5 vs. 30 mg·kg−one in puppies) also to The truth that
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